Destructive Pulse Syndrome © Andy D Kemp 2011/2018
The following theoretical article is based upon research and observations relating to my mother’s Motor Neuron Disease (towards answering some unexplained phenomena), the observation of my brother's life living with Multiple Sclerosis,and first hand knowledge in having Idiopathic Secondary Parkinson’s Disease.
All three of these illnesses are classed as being 'progressive' and ultimately have much in common, sharing similarities in neurological losses and or damage. Hence the term Destructive Pulse Syndrome has been adopted to define and focus upon the common symptoms of cell damage, the losses, deflection, and cross over of synapse patterns observed in PD, MND and MS; Towards furthering understanding in, coping with these diseases, treatment in Care, and the potential benefits found and witnessed through Exercise and certain Physiotherapy techniques, alleviating pressure within the spinal column and various regions throughout the body. (Written in the knowledge that not all cases of PD or other neurological conditions will be subject to identical physical damage as described.).
Understanding the aspects or concept of Destructive Pulse Syndrome (DPS), initially involves a very simplistic view of an energy circuit that relies upon feedback loops to govern its sustainability, before considering the complexities of individual cells, groups of cells, and how they may function or fail.
Imagine a simple self sustaining circuit system, comprising of a self governing pulse generator with a rechargeable battery and a wired in robot arm. Energy is sent from the generator around the circuit, the robot arm moves, burning off some of the energy and the remaining unused energy flows back into the generator, where more energy from the rechargeable battery amasses sufficient energy to send the next pulse to continue energising the arm. For such a self sustaining system to continually function, the pulse generator requires governors that balance and regulate all the available resources relevant to the whole system, so that the arm can retrieve 'food', an additional energy source to top up the rechargeable battery.
Now, if we put a single variable resistor into the circuitry (that interrupts the energy flow to and from the arm). This resistor inhibits the energy flow, and will not release the energy to activate the arm, until it has received an increased amount of pulsed energy (greater than the resistance), and likewise withhold more of the returning energy, eventually releasing a sufficiently large surge of energy back to the pulse generator, which due to the varible resistor's nature may be greater than energy the pulse generator normally supplies. Obviously, when this occurs, the pulse generator requires the means to adapt accordingly to address this imbalance, which may include the ability to, discharge or disperse the increased pulse of energy, and or an ability to replicate, replace any damaged or destroyed elements within the pulse generator; the living cell.
While the above is not a very scientific description, and the human body is much more complicated than a series of simple circuit cells all wired together. It may be useful when considering the full effects of internally damaged or scarred tissues within specific group types through out the body in and around the Peripheral nervous system, that may effectively kick start or perpetuate a variety of neurological disorders; dependant upon the location of damaged nerve tissue and the portions of the brain these collective increased returning synapse pulses then damage or destroy.
Exploring this idea or concept in Destructive Pulse Syndrome scenarios a little further; based upon the difference between, feeling well (or as fit as a fiddle) to the state in feeling under par or off colour. It occurs to me, to describe Nerve Synapses much in the same way as we consider or define Light; In having both aspects, in a Spectrum (wave form) and a quality of subsequent intensity.
Accordingly, the synapses themselves will be affected by, the states of hydration or dehydration, salts and biochemistry as well as the clarity (insulation and permeable properties) of the cells within the Peripheral Nervous System and ultimately the cells that form the Central Nervous System.
Similarly, as with light, there will also be a radiation aspect at work, as synapse 'Energy' physically bounces off tissue, as well as an effective responding increase or decrease of energy through the relevant reflective and absorption rates of adjacent biochemical gates it effectively stimulates.
Subsequently, any trapped rogue elements within individual or groups of cells will ultimately affect the 'Fizz' of life. Just as the interaction between the synapses within 'flesh tissue' and the synapses from Bacterial or Virus forms will ultimately deliver acceptable congenial interaction or unacceptable destructive interactions, that affect how we feel in regards to our health.
Equally, the interaction in synapse amongst the various groups of Bacteria within our airways and gut, will have a bearing upon the function of our organs and the effectiveness of our immune system; Pointing towards the importance in avoiding the creation of Environmental toxins that will effectively induce undesirable imbalances when particular bacteria are genetically modified, altered or displaced.
So the treatment of ills within the Peripheral Nervous system may point toward, Therapeutic Massage in Physiotherapy (to dislodge rogue and toxic elements) and Exercise (to assist the Lymphatic system) to eradicate DPS, with additional medical intervention to assist where necessary (or when conditions worsen) before such ills spread and manifest throughout the body and or within the Central Nervous System.
Akin to all this, is the other aspect of the Relax Datum Point (RDP) of each cell; The variable point, wherein a living cell is naturally at rest, seemingly inactive and or awaiting stimulation. A state in being where all the components are present and correct in their relevant format, and the RDP is fluctuating in keeping with the internal and external conditions. However, simply because a live cell is subject to changes the RDP can of course become completely out of sync, and may indeed be altered completely when a cell is damaged or physically altered in some form or another, to the detriment in the ability for satisfactory synapses and response.
For example: Altered or folded rogue proteins caused by heavy blows to groups or strings of cells arriving within a muscle tissue, may effectively become locked into place within those cells; When the muscle has automatically been activated for a prolonged period and then relaxed, before the offending rogue proteins have been expelled into the lymph system. In this scenario, the RDP completely alters, effectively including the rogue elements as a participant in the whole process in being, establishing 'a new norm' in that group of tissues. Which in turn will affect the physical 'elasticity' of the muscle cell and effectively the potency of all subsequent synapse, and ultimately perhaps mobility itself.
This in turn suggests, that by immediately treating the root cause’s (rogue proteins) with Physiotherapy including, gentle manipulation, stimulation (including target specific repair medicine), massage and sensibly good exercise, to remove rogue elements and reset the RDP, some aspects in progressive neurological diseases may be completely avoided, slowed down, or even over a period of time eventually cured.
While this may not cover every aspect or root causes of neurological deficits, even the genetic make up of nerve tissues may invite similar 'resistance and surge' synapse patterns occurring due to an altered cell wall structure. It is the approach towards, identifying and the treatment of the causes that is important, rather than just topping up any apparent deficiencies emanating from the causes.
In looking at this theory in more depth, we need to consider the finer details of biological electricity, or aspects of the synapse process in greater detail, and what happens within or around the damaged or compressed tissue cells (wall thickening, permability and density). How does the Nervous system actually function in its entirety?
In simplistic terms, the synapses or movement of energy through the nervous system will likely follow the easiest routes biochemically mapped out through the individual cell walls. While considering this, there is also the aspect of division or ‘doubling’ of a synapse within the nerve cells; where the command synapses propel a physical motion of the cell, while at the same time relaying a response synapse back to the brain, confirming the success or failure of commands.
In general terms, the aspects of breathing, heart beating, digestion etc., run subconsciously, while the motor aspects in moving the body through space, require a conscious thought process; and in both cases, the feedback synapses necessitate reception and response mechanisms within the brain, via the central and peripheral nerve system.
With damaged nerve cells however, the movement and energy value of the synapse will likely be hindered. The synapse may fire between cells at different locations in the cell walls, causing the current to divert or discharge on route, or sit within a cell until another incoming synapse provides sufficient power to enable the continuation of the synapses along the nerve fibre. The bi-fold nature of the synapse mechanism, means that nerve cells may allow command but withhold response, or vice versa, providing some undesired effects, that the brain will automatically try to re-address and or heal.
With entrapped nerve fibres or groups of nerve ganglion, where the cell walls are compacted together, the tissue will become denser. This may cause ‘bottle necking’ where the synapses are held within the cells, unable to continue their journey, until the external pressure releases, and the cell walls expand back to their operable density. With scarred tissue, the nerve cells may never recover or function as they did before.
Determining the extent of nerve damage in the various parts of the anatomy at the earliest stage possible is going to be key towards combating the knock on effects caused by insufficient or distorted synapse.
In re-describing DPS, the delay or failure of response synapses to reach the brain may cause the brain to send duplicated synapses, or choose neighbouring cells to dispatch new command synapses; leaving the first group of cells in a temporary idle mode. While a surge of response synapses returning to the brain may overwhelm or destroy vital cells.
This sort of brain damage may be temporarily healed as the human body is renewing cells all the time, although as we age the renewal processes begin to slow down.
As the muscles and nerve tissues lose the elasticity of youth, the damage from 'resistance and surge' synapse patterns are likely to physically increase; to the point where the original areas of damaged and hardened nerve fibres start to increase and give rise to adjacent or other associated areas of cells becoming affected in a similar fashion. In looking at this theory to identify DPS on a broader level,a common aspect of some injuries to the spinal column and central nerve, may relate directly to recognised neurological diseases and malfunctions.
For instance, DPS damage predominately caused or affected by damage in the upper areas of the spine and neck, may be attributed to a form of Motor Neurone Disease when the throat and swallowing mechanism are the significant areas initially malfunctioning; And in the later development stages of this disorder, the apparent mix-up in emotions, as the patient sheds tears when they are effectively laughing and vice versa (apparently laughing when actually being upset) are more easily understood, as the synapses are being redirected or disorientated along shared pathways and even self activating in a reverse mode due to concurrent RDP values.
The development of DPS through the upper regions of the spine may arise or stem from a variety of causes, such as; the development of curvatures due to muscle imbalance caused by hereditary genes, posture fixations caused through lifestyle or the working environment (such as in IT or office occupations where seating and screen positions remain at a constant), to accidents such as Whiplash injuries (associated with sharp breaking of a vehicle) or contact sports like rugby and boxing.
Likewise, the development of DPS through lower regions of the spine may cause similar symptoms attributed to Secondary or Idiopathic Parkinson’s where the synapses are hindered, disorientated and again routed along shared pathways; typically and often effecting one side of the body first, corresponding to the root of conspiring injuries. In cases associated with repetitive strain injuries, this may be more apparent in regard to the natural right or left handed aspect of the patient. Although, in spinal injuries, where dehydrated or disc hernias are present, it should be noted that injury, and or swelling on one side of the spine may cause ‘bottle necking’ scenarios on the opposite side, where the extending nerve branches are squeezed against the vertebrae bone structures around their corresponding areas of exit.
Another cause of DPS may emanate through the compacting of the spine at the base of the back, where dehydrated discs allow pressure to bear upon the sciatic nerves as they exit the spinal column. Noting that physical damage and scarring to the sciatic nerves may also occur, through a process of being constantly bruised by shocks associated with sports such as horse riding, prolonged periods in a static sitting posture taking the full weight of the body, and or with insufficient cushioning for the absorption of any mechanical vibration when operating machines.
DPS may also develop through physical limb injuries and areas of heavily scarred tissues, and where a major nerve has been damaged for life, and as a result, there is a continual ‘bottle necking’ as mentioned above. To a certain degree some of the nerve synapses may be re routed as the body compensates for the damaged nerve cells and the extent of such injuries may not become fully apparent until the central nervous system is directly effected, or the condition of the spinal column compounds the synapse imbalance with additional imbalances as a neurological disease progresses.
There are two important aspects that spring to mind through this theory. Firstly, where childhood injuries or abnormalities such as Spinal Senosis result in subsequent lack of growth in the areas of bone tissue through which nerves pass there will be continual effects upon Synapse from the ‘bottle necking’ and permanent scarring. Secondly, any ‘conditioning’ aspect set upon individual cells effected by the altered synapse patterns, may be reflected in their subsequent renewal, promoting a continual degeneration of the cell properties. This in turn underlines the necessity to ensure sufficient Physiotherapy and follow up procedures are put in place leading up to adulthood, more especially for spinal injury.
In the early stages, typical symptoms of DPS may include, pins and needle's, tingling and warmth feelings, current rushes and muscle twitching in a limb, a longer time period for a particular limb to achieve a relaxed state, and similar symptons as found in Restless Leg Syndrome.
Further symptoms may be seen as the body relaxes for sleep: These include, impromptu limb jerks, rushing of synapse charges up the central nerve resulting in momentary dizziness, drying and tingling in the wet areas of the mouth, echo in the ear chamber, buzzes and popping sensations in the brain. Such symptoms may only occur spasmodically during times of stress, or at times when the spinal discs are dehydrated sufficiently to magnify the resistance and surge aspect of the synapses.
Akin to this, any disorientation in a response synapse, reflecting the prior conditioning of the returning response synapses, occurring within the area of the brain, may result in mirrored misdirection due to the RDP or change in biochemistry. Producing feelings of anxiety, and other mental aspects, occasional illusion and hallucination, as the brain readapts to these fluctuations.
If you suffer from any of the symptons mentioned above please consult your family doctor or GP who will refer you to a Physiotherapist and a Neurologist if necessary.
Secondary Parkinson's Disease and DPS in the Gut.
Picking up upon the earlier point upon rogue proteins being locked within or into damaged muscle tissue, there is growing evidence to suggest that the gut wall may be affected sufficiently through Bacterial imbalances in such a way, that the patterns in synapses alter, effectively slowing down the digestion process to the extent of creating or causing portions of the intestine and colon to become 'leaky' which inturn may instill unwanted synapses via the Vagus nerve, while at the same time through changing the RDP in the cells of the gut wall towards establishing the rogue proteins as 'the new norm', with disasterous consequences.
Effectively the peristaltic wave becomes contributive and subjective to Destructive Pulse Syndrome through the breakdown in the defense mechanisms within the gut, and without early detection and repair, eventually manifests into conditions of a progressive disease or illness, such as Secondary or Idiopathic Parkinson's Disease, where the cells producing Dopamine diminish in number and or their ability to produce the relevant neuro transmitters.
Being part and parcel of Secondary Parkinson's Disease, the dopamine shortfalls and other effects caused by DPS are normally dealt with through the intake of synthetic dopamine and medicines as prescribed by the Neurologist.
Coping with, reducing and preventing DPS
Without doubt and most importantly, exercising and a well balanced diet, plus tuning your body to totally relax has to be good towards coping with any ill. While ensuring any losses in gut bacteria due to using medications like Antibiotics are restored as swiftly as possible may also assist.
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